Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. On the flip side, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes through the inhibition of FAK indicates elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by elevated pFAK localization in five-HT https://chancewkugr.dailyblogzz.com/32791338/considerations-to-know-about-pro33-login